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Order generic PhenerganAUTO-UPDATE `93 STAT KIT 600 AU693 ITEM # 121 104 103 * 810 1146 805 QTY 1 2 1 DESCRIPTION Aminophylline vial Ammonia Inhalants Amyl Nitrite Atropine, amp Atropine, PF Calcium Gluconate, vial Carpuject holder Clonidine, tablet D5W, 500ml bag Dexamethasone 5ml vial Dextrose, PF Diazepam, Carpuject Diphenhydramine, vial Epinephrine 1", PF Epinephrine, amp Furosemide Lasix, vial Ipecac Syrup Isuprel amp Lanoxin, amp Lidocaine, PF Narcan, amp Neo-Synephrine, amp Nitrostat Tabs, 25 ea. Nubain, amp Phenergan Promethazine, 25mg amp Procainamide, vial Propranolol, vial Sodium Bicarbonate, PF Verapamil, vial Laryngoscope Batteries, set of 2 Sutures, Vicryl Sutures, Prolene Plastic Seals. Nathan and Associates be predicated predominantly on the level of glycemic control achieved rather than on any other specific attributes of the intervention s ; used to achieve glycemic goals. The UKPDS compared three classes of glucose-lowering medications sulfonylurea, metformin, or insulin ; but was unable to demonstrate clear superiority of any one drug over the others with regard to complications 6, 7 ; . However, the different classes do have variable effectiveness in decreasing glycemic levels Table 1 ; , and the overarching principle in selecting a particular intervention will be its ability to achieve and maintain glycemic goals. In addition to the intention-to-treat analyses demonstrating the superiority of intensive versus conventional interventions, the DCCT and UKPDS demonstrated a strong correlation between mean A1C levels over time and the development and progression of retinopathy and nephropathy 23, 24 ; . Therefore, we think it is reasonable to judge and compare blood glucoselowering medications, and the combinations of such agents, primarily on the basis of the A1C levels that are achieved and on their specific side effects, tolerability, and expense. Nonglycemic effects of medications. In addition to variable effects on glycemia, specific effects of individual therapies on CVD risk factors, such as hypertension or dyslipidemia, were also considered important. We also included the effects of interventions that may benefit or worsen the prospects for long-term glycemic control in our recommendations. Examples of these would be changes in body mass, insulin resistance, or insulin secretory capacity in type 2 diabetic patients. Choosing specific diabetes interventions and their roles in treating type 2 diabetes Numerous reviews have focused on the characteristics of the specific diabetes interventions listed below 2533 ; . The aim here is to provide enough information to justify the choices of medications, the order in which they are recommended, and the utility of combinations of therapies. Unfortunately, there is a dearth of highquality studies that provide head-to-head comparisons of the ability of the medications to achieve the currently recommended glycemic levels. The authors highly recommend that such studies be conducted. However, even in the absence of rigorous, comprehensive studies that directly compare the efficacy of all availDIABETES CARE, VOLUME 29, NUMBER 8, AUGUST 2006. Phenergan pregnancy682 Non-isotope-based quantitative proteomics in the absence of genomic sequence information. Panisko, E.A.1, Daly, D.S.2, Anderson, K.K.2 and Baker, S.1 1Fungal Biotechnology, Pacific Northwest National Laboratory, Richland, WA, USA. 2Conputational and Information Sciences, Pacific Northwest National Laboratory, Richland, WA, USA. Mass spectrometry MS ; based proteome analysis generates an enormous amount of data in the form of a "snapshot" of peptides present in a sample and is therefore an attractive platform for discovery and assessment of protein-based signatures of environmental change. One major hurdle for the implementation of MS based proteomic analysis of environmental samples is a paucity of genomic sequence information for organisms that may be present in a given sample. Another complicating factor derives from the fact that most quantitative proteomics strategies rely on differential isotope labeling of samples. We are developing a proteomic fingerprinting method for complex environmental samples that does not require genomic sequence for the organisms present. Instead this method relies on a comprehensive generic peptide sequence database, rigorous experimental design and statistical analysis. We are validating the system using two strains of a single organism, Trichoderma reesei. Because the T. reesei genome has been sequenced we can run a nongenome sequence based analysis and compare validate our findings with a genome sequence based analysis. Following validation using T. reesei, we will apply our method to complex, multi-organism environmental samples and claritin. Phenergan cure6.5.13 Drugs to avoid in prophylactic intervention and periactin. Received 3 10 2005; revised 8 17 2005; accepted 9 1 2005. Grant support: Grants-in-aid for scientific research from the Ministry of Education, Culture, Sports, Science and Technology of Japan. The costs of publication of this article were defrayed in part by the payment of page charges. This article must therefore be hereby marked advertisement in accordance with 18 U.S.C. Section 1734 solely to indicate this fact. We thank Dr. Isao Kanatani, Tomoko Matsushita and Chie Hagihara for their valuable technical assistance. Crebelli R, Benigni R, Franekic J, et al. 1988. Induction of chromosome malsegregation by halogenated organic solvents in Aspergillus-nidulans unspecific of specific mechanism Mutat Res 201: 401-412. * Crebelli R, Conti G, Conti L, et al. 1984. Induction of somatic segregation by halogenated aliphatic hydrocarbons in Aspergillus nidulans. Mutat Res 138: 33-38. * Crespi CL, Seixas GM, Turner TR, et al. 1985. Mutagenicity of 1, 2-dichloroethane and 1, 2-dibromoethane in two human lymphoblastoid cell lines. Mutat Res 142: 133-140. Crisp TM, Clegg ED, Cooper RL, et al. 1998. Environmental endocrine disruption: An effects assessment and analysis. Environ Health Perspect 106 Suppl. 1 ; : 11-56. * Croen LA, Shaw GM, Sanbonmatsu L, et al. 1997. Maternal residential proximity to hazardous waste sites and risk for selected congenital malformations. Epidemiology 8 4 ; : 347-354. Cronin MTD. 1996. Quantitative structure-activity relationship QSAR ; analysis of the acute sublethal neurotoxicity of solvents. Toxicol in Vitro 10: 103-110. Crume RV. 1991. The comparison of health risks between different environmental media at Superfund hazardous waste sites. Proc Ann Meet Air Waste Manage Assoc 84: 91 109.4. Dacre JC. 1994. Hazard evaluation of army compounds in the environment. Drug Metab Rev 26: 649662. * Daft J. 1987. Determining multifumigants in whole grains and legumes, milled and low-fat grain products, spices, citrus fruit, and beverages. J Assoc Off Anal Chem 70: 734-739. * Daft JL. 1988. Rapid determination of fumigant and industrial chemical residues in food. J Assoc Off Anal Chem 71: 748-760 and entocort. Ondansetron zofran ; promethazine phenergan ; prochlorperazine compazine ; metoclopramide reglan ; trimethobenzamide tigan ; 1 2 3 « previous page glossary next page » next: next steps » printer-friendly format email to a friend privacy policy women's health find out what women really need. Mug of the Month This month the "Mug of the Month" is awarded to pharmacist Janet Walker for alerting Detective Hodges to Carrie L. Clemmons 24 year old black female ; , Lee E. Osborne 28 year old black male ; and James M. Neal Jr. 27 year old black male ; . This dubious trio was arrested for presenting multiple stolen forged prescriptions from Norton Hospital for Phenergan VC w Codeine. The trio also had notes listing the DEA numbers of many Metro Louisville physicians. The only notes these three will now be taking are how many days they have to serve in the beautiful confines of the Louisville Metro Jail. Thank you Janet and please keep those tips coming and zaditor. Side effects of PhenerganThe pharmacological actions of bisphosphonates are due to the inhibitory effects on bone resorption, but little is known about the bisphosphonate action on bone formation. The purpose of this study is to elucidate the actions of bisphosphonates, clodronate, on bone formation in the experimental in vivo and in vitro rat models. The bone mineral density BMD ; was decreased in the rats fed a low-calcium diet 0.05% Ca ; for 6 days compared with the rats fed a normal-calcium diet 0.5% Ca ; . The decrease in BMD was suppressed in the 2 mgP day and the 4 mgP day clodronate administrations. Bone formation rate BFR ; in rats fed a low-calcium diet was significantly increased compared with the rats fed a normal-calcium diet, and the 2 mgP clodronate administration further increased the BFR. In the cultured rat bone marrow cells, the area of mineralized nodules was significantly increased at 10-7 and 10-6 M clodronate, but high concentration of clodronate decreased the area. From these results, it is concluded that clodronate stimulates bone formation when the drug was given to a rat with a relatively lower dose that is sufficient to prevent bone resorption and that this effect may be due to the stimulatory effect on the differentiation process of osteoblasts and zyrtec and Order phenergan. DECISION AND ORDER Pacific Employers Insurance Company Carrier ; challenged the decision of the Medical Review Division MRD ; of the Texas Workers' Compensation Commission Commission ; ordering the Carrier to pay for certain medications for Claimant ; for the period December 28, 2002, through April 10, 2003. The MRD concluded that the medications were medically necessary to treat Claimant. During the disputed period, Claimant had paid for the medications herself as Carrier had ceased reimbursing Claimant's pharmacy for the drugs at issue before December 28, 2002. Based on the evidence, Carrier failed to meet its burden of proof to show that some of the medications prescribed for Claimant were not medically necessary, specifically it failed to demonstrate that Zanaflex Tizanidine ; , Bextra, Clonazepam Klonopin ; , and Lidoderm patches were not needed to properly treat Claimant. Carrier should reimburse Claimant the amounts she paid for these medications purchased between December 28, 2002, and April 10, 2003; a total of 2.16. However, Carrier met its burden of proof to demonstrate there was no medical necessity for the other medications prescribed, specifically the following: Keratine cream Keta Neur Clon Bac Compound ; , Zonalon cream, Norflex, MS Contin morphine ; , Detromethorphan, Lasix, Phenergan Promethazine ; , Imitrex, Tegaderm w Lidoderm, Miralax powder, Famotidine, and Senokot. The hearing in this matter convened on March 31, 2004, in Austin, Texas, with Administrative Law Judge ALJ ; Cassandra Church presiding. The ALJ closed the record on April 23, 2004, upon receipt of the parties' closing arguments. Claimant appeared on her own behalf, assisted by Juan Mireles of the Commission's Ombudsman Office. Carrier was represented by Laurie Gallagher, attorney. The Commission did not participate in the hearing. Do not take Phenergan if you are pregnant or plan to become pregnant. It may affect your developing baby if you take it during pregnancy. Your pharmacist or doctor will discuss the benefits and possible risks of taking the medicine during pregnancy. Do not take Phenergan if you are breastfeeding or plan to breastfeed. It passes into the breast milk and there is a possibility that the baby may be affected. Do not take Phenergan after the expiry date EXP ; printed on the pack. If you take this medicine after the expiry date has passed, it may not work as well. Do not take Phenergan if the packaging is torn or shows signs of tampering and singulair. Found that adverse effects were the most common reason 35% ; .8 Data from PA-PSRS show that 62% of medication-related falls that result in a Serious Event affected the elderly. In 1991, 13 nationally recognized experts in geriatrics reached a consensus on explicit criteria for certain medications that may lead to ADEs and were considered to be inappropriate for use in nursing home patients. These criteria were originally developed by Dr. Mark Beers and are commonly referred to as the "Beers Criteria." The criteria, most recently updated in 2003, 9, 10 are based on the risk-benefit definition of appropriateness, meaning that the use of a medication is considered to be appropriate if its use has potential benefits that outweigh potential risks.11 The Beers criteria define three categories of drug use or selection that are inappropriate for elderly patients. The categories, along with some examples are: 1. Inappropriate drug choice, i.e., medications generally to be avoided in the elderly population. Examples include: a ; Long-acting benzodiazepines, including diazepam VALIUM ; , flurazepam DALMANE ; , and chlordiazepoxide LIBRIUM ; which have long half-lives. This can lead to accumulation of the drug, leading to excessive sedation and an increase in the risk of falls and fractures. b ; Meperidine DEMEROL ; , which can cause confusion and its metabolites can lead to seizures. c ; Anticholinergics and antihistamines, including diphenhydramine BENADRYL ; , chlorpheniramine CHLORTRIMETON ; , hydroxyzine ATARAX, VISTARIL ; and promethazine PHENERGAN ; . These agents have potent anticholinergic effects and cause confusion and sedation. Diphenhydramine may be used in the lowest effective dose and only for emergency treatment of allergic reactions. 2. Excess dosage, i.e., medications at a dose or duration of therapy not to be exceeded. Examples include: a ; Long-term use of stimulant laxatives such as bisacodyl DULCOLAX ; and cascara sagrada, which may be appropriate in the presence of opiate analgesic use, but may exacerbate bowel dysfunction. b ; Doses for digoxin LANOXIN ; should not exceed 0.125 mg day except when treating. Phenergan mg po qid prn follow-up: or for signs of dehydration, abdominal pain, failuretoimprove, development of fever, or bloody diarrhea or emesis. 58 days ago in the clubs fleet already consists of a porsche carrera · authority: 31 but it was phenergan and child that s what her father site oilstarwhat 2008 06 03 but-it-was-phenergan-and… but it was phenergan and child that s what her father june 3rd, 2008 by normankarwowski hindu nationalist bjp wins state elections in southern india for. Some medicines and phenergan may interfere with each other. Emcyt Ergamisol Eulexin Fludara Hexalen Hydrea Leukeran Lupron Lysodren Matulane Myleran Nolvadex Purinethol Sandostatin Teslac Thioguanine Uracil Mustard VePesid CHOLESTEROL LOWERING Lower Cost Generics cholestiramine resin gemfibrozil Brands Lescol Lipitor Niaspan Pravachol Prevalite COUGH & COLD MEDICATIONS Lower Cost Generics codeine quaifenesin codeine pseudoephedrine quaifenesin dextromethropan promethazine quaifenesin hydrocodone quaifenesin pseudoephedrine ext-rel Brands Narcotic Containing Products Entuss-D Phenergan DM Tussionex Suspension Non-Narcotic Containing Products Entex Phenegran DM Poly-Histine DM, D, Ped. Only prescription cough and cold drugs are on the formulary. The use of overthe-counter products is recommended when possible. DIABETES Lower Cost Generics glipizide glyburide Brands Amaryl Avandia Glucophage Glucotrol XL and buy claritin. WARNING: Many antitoxins are made from horse serum, such as some tetanus antitoxins and the antivenoms for snakebite and scorpion sting. With these there is a risk of causing a dangerous allergic reaction allergic shock, see p. 70 ; . Before you inject a horse serum antitoxin, always have epinephrine ready in case of an emergency. In persons who are allergic, or who have been given any kind of antitoxin made of horse serum before, it is a good idea to inject an antihistamine like promethazine Phenergan ; or diphenhydramine Benadryl ; 15 minutes before giving the antitoxin. Scorpion antitoxin or antivenom Name: price: for Often comes lyophilized in powdered form ; for injection Different antivenoms are produced for scorpion sting in different parts of the world. In Mexico, Laboratories BIOCLON produces Alacramyn. Antivenoms for scorpion sting should be used only in those areas where there are dangerous or deadly kinds of scorpions. Antivenoms are usually needed only when a small child is stung, especially if stung on the main upper part of the body or head. To do most good, the antivenom should be injected as soon as possible after the child has been stung. Antivenoms usually come with full instructions. Follow them carefully. Small children often need more antivenom than larger children. Two or 3 vials may be necessary. Most scorpions are not dangerous to adults. Because the antivenom itself has some danger in its use, it is usually better not to give it to adults. Snakebite antivenom or antitoxin Name: price: for Often comes in: bottles or kits for injection Antivenoms, or medicines that protect the body against poisons, have been developed for the bites of poisonous snakes in many parts of the world. If you live where people are sometimes bitten or killed by poisonous snakes, find out what antivenoms are available, get them ahead of time, and keep them on hand. Some antivenoms--those in dried or `lyophilized' form--can be kept without refrigeration. Others need to be kept cold. The following are distributors of antivenom products in different parts of the world. In many countries, antivenoms are available through the government: North America: Crofab TM ; Crotalidae Polyvalent Immune Fab-Ovine ; for rattlesnakes, copperheads, cottonmouths, and water moccasins. From Fougera, Inc., 1-800-645-9833, fougera . Product information also from the manufacturer, Protherics, 1-800-231-0206, or 1-615-963-4528, e-mail: information protherics , protherics products antibody Mexico, Central America, and South America: Antivipmyn and Antivipmyn tri Faboterapia polivalente antiviperino ; for rattlesnakes and other pit vipers, as well as nauyaca, terciopelo, mapana, toboba, jararaca, cuatro narices, cola de hueso, barba amarilla, palanca, and others. From Instituto Bioclon, Mexico, D.F., tel: 52 ; 5575-0070, 52 ; 5575-4016, or 1-800-021-6887, bioclon .mx Antivenoms are also available from Instituto Clodomiro Picado, Facultad de Microbiologia, Universidad de Costa Rica, San Jose, Costa Rica: icp.ucr.ac.cr, and Instituto Butantan, Sao Paulo, Brazil, tel: 011 ; 3726-7222, fax: 011 ; 3726-1505, email: instituto butantan.gov , butantan.gov Africa: Polyvalent antivenoms for puff adder, Gaboen viper, green mamba, Jameson's mamba, black mamba, cape cobra, forest cobra, snouted cobra and Mozambique spitting cobra. From South Africa Vaccine Producers PTY Ltd., P.O. Box 28999, Sandringham 2131, South Africa, tel: 27-11-386-6000, fax: 27-11-386-6016, savpo India: Antivenoms for Indian cobra, Indian krait, Russell's viper, Saw-scaled viper and others, from: Haffkine Biopharmaceutical Co., Bombay, India, tel: 91- 22-412-9320 22, fax: 91-22-416-8578, vaccinehaffkine . Central Research Institute of Kasuli, Kasuli, India, tel: 01-792-72114, fax: 0- 792-72016. Serum Institute of India, tel: 91-20-269-93900, fax: 91-20-269-93921, seruminstitute . Indonesia: Biofarma, Bandung, Indonesia, tel: 022-233-755, fax: 022-204-1306, biofarma.co.id. Thailand: Thai Red Cross Society, Bangkok, Thailand, tel: 66-2255-461, fax: 66-2252-7795, redcross.or.th. Instructions for the use of snakebite antivenoms usually come with the kit. Study them before you need to use them. The bigger the snake, or the smaller the person, the larger the amount of antivenom needed. Often 2 or more vials are necessary. To be most helpful, antivenom should be injected as soon as possible after the bite. Be sure to take the necessary precautions to avoid allergic shock see p. 70. Functioning prepared for the IMMPACT-II consensus meeting Kerns, 2003 ; and discussions among the participants, the Beck Depression Inventory BDI; Beck et al., 1961 ; and the Profile of Mood States POMS; McNair et al., 1971 ; are recommended as core outcome measures of emotional functioning in chronic pain clinical trials. Both the BDI and POMS have well-established reliability and validity in the assessment of symptoms of depression and emotional distress, and they have been used in numerous clinical trials in psychiatry and in an increasing number of chronic pain clinical trials Kerns, 2003 ; . In research in psychiatry and in chronic pain, the BDI provides a wellaccepted measure of the level of depressed mood in a sample and its response to treatment. The POMS assesses six mood states--tensionanxiety, depressiondejection, angerhostility, vigoractivity, fatigueinertia, and confusionbewilderment--and also provides a summary measure of total mood disturbance. Although the discriminant validity of the POMS scales in patients with chronic pain has not been adequately documented, the POMS has scales for the three most important dimensions of emotional functioning in chronic pain patients depression, anxiety, anger ; and also assesses three other dimensions that are very relevant to chronic pain and its treatment, including a positive mood scale of vigor activity. Moreover, the POMS has demonstrated beneficial effects of treatment in some but not all ; recent chronic pain trials e.g. Rowbotham et al., 1998 ; . For these reasons, administration of both the BDI and the POMS is recommended in chronic pain clinical trials to assess the major aspects of the emotional functioning outcome domain. The assessment of emotional functioning in patients with chronic pain presents a challenge because various symptoms of depression--such as decreased libido, appetite or weight changes, fatigue, and memory and concentration deficits--are also commonly believed to be consequences of chronic pain and the medications used for its treatment Gallagher and Verma, 2004 ; . It is unclear whether the presence of such symptoms in patients with chronic pain and other medical disorders ; should nevertheless be considered evidence of depressed mood, or whether the assessment of mood in these patients should emphasize symptoms that are less likely to be secondary to physical disorders Wilson et al., 2001 ; . Because the evidence indicates that measures of emotional functioning are adequately reliable, valid, and responsive when used in the medically ill Kerns, 2003 ; , the authors recommend that the principal analyses of the BDI and POMS in chronic pain clinical trials use the original versions without adjustment for presumed confounding by somatic symptoms. Depending on the specific objective of the clinical trial, supplemental analyses could be conducted to separately examine non-somatic and somatic aspects of emotional functioning. If your doctor or pharmacist tells you to stop taking phenergan or it has passed the expiry date, ask your pharmacist what to do with any that are left over. Phenergan therapy391 June 2000 Scientific Affairs - 9 SCREENING AND EARLY DETECTION OF PROSTATE CANCER RESOLUTION 511, I-99 ; HOUSE ACTION: RECOMMENDATIONS ADOPTED AS FOLLOWS IN LIEU OF RESOLUTION 511 I-99 ; AND RESOLUTION 517 AND REMAINDER OF REPORT FILED Resolution 511, introduced by the American Urological Association and the American Association of Clinical Urologists and referred to the Board of Trustees at the 1999 Interim Meeting asks: That the American Medical Association adopt the American Cancer Society and American Urological Association policy on early detection of prostate cancer that "both prostate specific antigen PSA ; and digital rectal examination DRE ; should be offered annually, beginning at age 50 years, to men who have at least a 10-year life expectancy, and to younger men who are at high risk." METHODS Published studies from the years 1988 to March 2000 were identified through MEDLINE and Lexis Nexis Medical Library searches for English-language articles using the key words "prostatic neoplasms, " "palpation, " "prostatespecific antigen, " and "mass screening, " cross-indexed with the additional MeSH terms "prevention and control, " "tumor markers, " "sensitivity and specificity, " and "diagnosis." A total of 234 articles were retrieved for analysis. Additional articles were identified by review of the references cited in these publications. INTRODUCTION Prostate cancer has become the most common type of cancer among men, and is the second leading cause of cancer deaths in males. However, because it occurs primarily in older men, prostate cancer is the 21st leading cause of years of life lost. In 1999, approximately 179, 300 men in the United States were diagnosed with prostate cancer and approximately 37, 000 deaths were attributed to this disease. The lifetime risk for clinical prostate cancer among men in the United States is approximately 10%; approximately 3% die of this disease. Major risk factors are age, family history, race, and possibly dietary fat. Autopsy and cystoprostatectomy studies have shown a prostate cancer prevalence of 30% to 46% based on histologic examination. For men over age 50 years, the weighted average of prostate cancer prevalence based on autopsy studies is 30%. Approximately 20% to 25% of these cancers are believed to be clinically significant. Thus, the risk for a 50-year-old man with a 25-year life expectancy of having microscopic cancer is approximately 30%, of having clinically evident disease 10%, and of dying from prostate cancer 3%. The disparity between the 30% prevalence of histologic prostate cancer in men older than 50 years of age and the 3% lifetime risk of death, shows the difficulty in distinguishing cancer that is destined to cause illness and death, from indolent disease. Nevertheless, early detection is potentially important because the survival of patients diagnosed in early-stage disease is substantially better than that of patients diagnosed with late-stage disease. Patients with symptomatic disease generally have late-stage cancer that has spread and is incurable. The main screening tests in use today for the early detection of prostate cancer are digital rectal examination DRE ; and measurement of the serum concentration of prostate-specific antigen PSA ; . PSA is a glycoprotein with serine protease activity produced primarily by epithelial cells lining the acini and ducts of the prostate gland. PSA is secreted into the lumina of the prostatic ducts and is present in high concentrations in seminal fluid. Plasma concentrations are normally low but are increased by conditions that disrupt normal prostate structure and function ie, inflammation, infection, hyperplasia, primary and metastatic cancer ; . Androgens regulate expression of the PSA gene. The Food and Drug Administration FDA ; approved the PSA test in 1986 to monitor disease status of patients with prostate cancer, and in 1994 to aid in the detection of prostate cancer in men aged 50 years and older. Use of the PSA test for the diagnosis of prostate cancer, either in response to symptoms or for screening, increased dramatically beginning in 1988. Medic946 , i can safely tell you that phenergan not only potentiates ms but other opiates such as dilaudid as well. Cheap Phenergan onlineBuy generic PhenerganPhejergan, pjenergan, phenerggan, phenergna, phenerrgan, phdnergan, phenetgan, phenergab, ph4nergan, phensrgan, phenergaan, phenergxn, phenergann, henergan, pheneggan, phenergwn, phene4gan, pnenergan, puenergan, pgenergan, phemergan, phenergaj, phenerga, phenergam, phenedgan, pheergan, pehnergan, ohenergan, phenerban, phenerhan, phenefgan, phenrgan, phenergah, phenergzn, phenergn, phen4rgan, phhenergan. |
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